Allergies and Types of Allergic Reactions in Children

1. Mahalingam Jagadeeshwaran

2. Stephen Priyadharshini

3. Arundhathi Shaji

4. Osmonova Gulnaz

(1. Student, International Medical Faculty, Osh State University, Osh, Kyrgyz Republic)

(2. Student, International Medical Faculty, Osh State University, Osh, Kyrgyz Republic)

(3. Student, International Medical Faculty, Osh State University, Osh, Kyrgyz Republic)

(4. Teacher, International Medical Faculty, Osh State University, Osh, Kyrgyz Republic)

 

Abstract

Allergic disorders are among the most common chronic conditions affecting children, resulting from exaggerated immune responses to otherwise harmless environmental or dietary substances. These reactions are primarily mediated by immunological mechanisms, most notably immunoglobulin E (IgE), leading to the release of inflammatory mediators such as histamine from mast cells and basophils. Allergic reactions are broadly classified into four types based on the Gell and Coombs classification, with Type I (immediate hypersensitivity) being the most clinically relevant in pediatric populations. This category includes conditions such as urticaria, angioedema, and anaphylaxis, which vary in severity from mild cutaneous manifestations to life-threatening systemic emergencies.

Anaphylaxis represents the most severe form of allergic reaction, characterized by rapid onset and involvement of multiple organ systems, including the skin, respiratory, cardiovascular, and gastrointestinal systems. Prompt recognition and immediate administration of intramuscular epinephrine are critical to prevent fatal outcomes. Urticaria, commonly known as hives, presents as transient, pruritic wheals on the skin and is often triggered by infections, foods, or medications. Angioedema, or Quincke’s edema, involves deeper dermal and subcutaneous swelling, frequently affecting the face and airway, and may occur independently or in association with urticaria and anaphylaxis.

This article provides a comprehensive overview of allergies in children, detailing the classification of allergic reactions, underlying pathophysiology, clinical manifestations, and management strategies, with particular emphasis on anaphylaxis, urticaria, and angioedema. Early diagnosis, identification of triggers, and appropriate therapeutic interventions are essential in reducing morbidity and preventing complications in pediatric patients.

Introduction to Allergies

Allergies are exaggerated immune responses that occur when the body reacts to substances (allergens) that are usually harmless to most individuals. In children, allergic diseases are particularly common due to the developing immune system and genetic predisposition. When a child with an allergic tendency is exposed to an allergen such as food proteins, pollen, dust mites, or insect venom, the immune system mistakenly identifies it as a threat and produces immunoglobulin E (IgE) antibodies. These antibodies bind to mast cells and basophils, leading to the release of chemical mediators such as histamine, leukotrienes, and prostaglandins. These mediators are responsible for the clinical symptoms of allergy, which can range from mild skin reactions to life-threatening systemic responses.

Allergic disorders in children include conditions such as asthma, allergic rhinitis, atopic dermatitis, urticaria, angioedema, and anaphylaxis. The prevalence of allergies has been increasing worldwide, possibly due to environmental factors, lifestyle changes, and reduced exposure to microbes in early life (hygiene hypothesis). Early recognition and appropriate management are essential to prevent complications and improve quality of life.

Types of Allergic Reactions

Allergic reactions are classified based on the underlying immunological mechanism. The most widely accepted classification is the Gell and Coombs classification, which divides allergic reactions into four types.

Type I Hypersensitivity (Immediate Reaction)

Type I hypersensitivity is the most common form of allergic reaction in children and is mediated by IgE antibodies. Upon first exposure to an allergen, the child becomes sensitized, and IgE antibodies are produced. On subsequent exposure, the allergen binds to IgE on mast cells, causing degranulation and release of histamine and other mediators. This leads to symptoms that occur within minutes to hours.

Common examples in children include:

Anaphylaxis, Urticaria (hives), Angioedema, Allergic rhinitis, Bronchial asthma.

These reactions are rapid in onset and can vary in severity from mild itching to severe airway obstruction.

Type II Hypersensitivity (Cytotoxic Reaction)

Type II reactions involve IgG or IgM antibodies directed against antigens on the surface of cells. This leads to cell destruction through complement activation or phagocytosis. Although less common in typical pediatric allergies, these reactions can occur in certain drug allergies or autoimmune conditions.

Examples include: Hemolytic anemia, Transfusion reactions

Type III Hypersensitivity (Immune Complex Reaction)

In Type III reactions, antigen-antibody complexes are formed and deposited in tissues, leading to inflammation and tissue damage. These reactions are less commonly seen as classical allergies in children but may occur in certain conditions.

Examples include: Serum sickness, Some drug reactions

Type IV Hypersensitivity (Delayed Reaction)

  • Type IV reactions are mediated by T lymphocytes rather than antibodies. These reactions occur hours to days after exposure to the allergen. They are commonly seen in contact allergies.

  • Examples include:

  • Contact dermatitis (e.g., reaction to metals, cosmetics)

  • Tuberculin skin test reaction

Anaphylaxis in Children

Definition and Overview

Anaphylaxis is a severe, life-threatening systemic allergic reaction that occurs rapidly after exposure to an allergen. It involves multiple organ systems, particularly the skin, respiratory system, cardiovascular system, and gastrointestinal tract. In children, common triggers include food allergens (such as peanuts, milk, eggs), insect stings, medications, and latex.

Pathophysiology

Anaphylaxis is primarily an IgE-mediated Type I hypersensitivity reaction. Upon exposure to the allergen, massive degranulation of mast cells and basophils occurs, releasing large amounts of histamine and other mediators. This leads to:

Vasodilation and increased vascular permeability → hypotension and shock

Bronchoconstriction → breathing difficulty

Mucosal edema → airway obstruction

Clinical Features

The symptoms of anaphylaxis usually appear within minutes and progress rapidly. They may include:

  • Skin manifestations: urticaria, itching, flushing

  • Respiratory symptoms: wheezing, stridor, shortness of breath

  • Cardiovascular signs: hypotension, tachycardia, collapse

  • Gastrointestinal symptoms: vomiting, abdominal pain, diarrhea

  • In severe cases, it can lead to anaphylactic shock, which is a medical emergency.

Management

Immediate treatment is crucial in anaphylaxis. The primary steps include:

  • Intramuscular adrenaline (epinephrine) – first-line treatment

  • Airway management and oxygen support

  • Intravenous fluids for hypotension

  • Antihistamines and corticosteroids as adjunct therapy

  • Children at risk should carry an epinephrine auto-injector and caregivers must be trained in its use.

Urticaria (Hives) in Children

Definition

Urticaria is a common allergic condition characterized by the sudden appearance of raised, itchy, red or pale swellings on the skin called wheals. These lesions can vary in size and shape and may appear anywhere on the body.

Etiology

  • Urticaria in children can be triggered by various factors, including:

  • Food allergens (milk, eggs, nuts)

  • Medications (antibiotics, NSAIDs)

  • Infections (viral infections are a common cause in children)

  • Physical stimuli (cold, heat, pressure)

  • Insect bites

  • In many cases, the exact cause may not be identified.

Pathophysiology

Urticaria results from the release of histamine and other mediators from mast cells in the skin. This leads to increased permeability of blood vessels, causing localized swelling and redness.

Clinical Features

  • Raised, itchy wheals with a central pale area

  • Lesions may change shape and location rapidly

  • Each lesion usually lasts less than 24 hours

  • May be associated with angioedema

Types of Urticaria

Acute urticaria: lasts less than 6 weeks

Chronic urticaria: persists for more than 6 weeks

Management

  • Identification and avoidance of triggers

  • Antihistamines are the mainstay of treatment

  • Severe cases may require corticosteroids

Angioedema (Quincke’s Edema) in Children

Definition

Angioedema is a deeper form of swelling that occurs in the dermis and subcutaneous tissues, often affecting the face, lips, eyelids, tongue, and throat. It is sometimes referred to as Quincke’s edema.

Etiology

Angioedema may occur due to:

  • Allergic reactions (IgE-mediated)

  • Medications (e.g., ACE inhibitors)

  • Hereditary causes (C1 esterase inhibitor deficiency)

  • Idiopathic causes

Pathophysiology

Like urticaria, angioedema involves increased vascular permeability due to mediator release. However, it affects deeper layers of the skin and mucosa, leading to more pronounced swelling.

Clinical Features

Sudden, non-pitting swelling of the skin

Commonly affects lips, eyelids, and tongue

May involve airway. Usually not itchy, but may be painful or tense

Complications

The most serious complication is airway obstruction, especially when the tongue or larynx is involved. This can lead to respiratory distress and requires emergency intervention.

Management

Antihistamines for mild cases, Corticosteroids for moderate to severe cases, Epinephrine if associated with anaphylaxis, Airway management in severe cases.

Conclusion

Allergic reactions in children range from mild conditions like urticaria to severe, life-threatening emergencies such as anaphylaxis. Understanding the types of hypersensitivity reactions and recognizing early symptoms are crucial for timely diagnosis and treatment. Urticaria and angioedema are common manifestations that may occur independently or as part of a systemic allergic response. Anaphylaxis requires immediate medical attention and prompt administration of epinephrine to prevent fatal outcomes. Proper education of caregivers, avoidance of known allergens, and preparedness for emergencies are essential components of managing pediatric allergies effectively.

References

· Nelson Textbook of Pediatrics. Kliegman RM, St. Geme JW, Blum NJ, Shah SS, Tasker RC, Wilson KM. Nelson Textbook of Pediatrics. 21st ed. Philadelphia: Elsevier; 2020.

· Middleton's Allergy: Principles and Practice. Adkinson NF, Bochner BS, Burks AW, et al. Middleton's Allergy: Principles and Practice. 9th ed. Elsevier; 2020.

· World Health Organization. Allergic diseases and asthma guidelines. Available from: https://www.who.int⁠

· American Academy of Pediatrics. Clinical reports and guidelines on pediatric allergies and anaphylaxis. Available from: https://www.aap.org⁠

· World Allergy Organization. Anaphylaxis guidelines and allergy resources. Available from: https://www.worldallergy.org⁠

· American Academy of Allergy, Asthma & Immunology. Practice parameters for allergy and immunology. Available from: https://www.aaaai.org⁠

· Robbins and Cotran Pathologic Basis of Disease. Kumar V, Abbas AK, Aster JC. Robbins and Cotran Pathologic Basis of Disease. 10th ed. Elsevier; 2021.

· Oxford Handbook of Paediatrics. Lissauer T, Carroll W. Oxford Handbook of Paediatrics. 3rd ed. Oxford University Press; 2018

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